The country has had an epidemic of infections with fungi that is threatening public health.
But, as the country’s leading infectious disease expert told me, you can’t simply fix the fungus problem.
Instead, you have to find new ways to help people manage their own infections.
A year ago, I began my first major public-health assignment as the director of the National Center for Emerging and Zoonotic Infectious Diseases at the National Institutes of Health, a post that has since evolved into a fellowship.
For the past two years, I’ve been leading a team of researchers that has been looking at ways to combat the fungus epidemic.
The new work we’re doing is focused on understanding the mechanisms that allow us to fight infections, such as the roles played by the body’s immune system, gut microbiome, and microbiome-derived molecules, or MDRs, in the pathogenesis of the infection.
And this work could help us devise new treatments to fight the spread of the fungus.
This new work has a huge impact on the way we think about the infection, how we treat it, and how we prevent the disease.
Fungus is a complex organism that infects us through direct contact, through the skin and through our mouths.
Its spread is so widespread that it can take years for symptoms to appear.
The virus usually causes only mild symptoms, but when symptoms become severe, the virus can lead to severe illness, including death.
But when people become infected with the fungus, they can have the same disease, or worse.
Our understanding of the role of the microbiome and MDR is evolving.
The microbes that live inside us are changing as we age.
Our bodies are adapting to the way they’re being infected by the fungus as we get older.
In other words, we are changing our microbiome in ways that affect our health.
This means that our immune system and microbiome are playing a critical role in the disease process, because they can play a crucial role in preventing the fungus from spreading.
We’ve been working on a number of fronts with our team to understand the microbiome’s role in controlling the disease, including the microbiome-specific MDR molecule, known as TLR5, that’s a protein that’s released from the gut after we eat certain foods, including certain foods containing the fungus-causing fungus species Candida albicans.
TLR1, which is the receptor that normally recognizes the TLR4 protein, has been shown to play a role in immune system responses to the fungi.
The TLR2 protein, known to have been released by the microbes, has also been shown in some studies to be involved in the ability of fungi to evade the immune system.
But TLR3 has been the one area where we haven’t found a strong correlation between TLR expression and infection severity.
TLRs are also involved in other immune system-mediated reactions, including inflammation, and inflammation is associated with the development of some chronic diseases, such chronic obstructive pulmonary disease.
The fact that TLRs play such a big role in regulating inflammation is what makes them a particularly useful tool in fighting the infection; inflammation has been linked to the development and progression of many chronic diseases.
I want to focus on TLR and its role in how we fight the infection and how to treat it.
And that is because we know that the microbiome plays a key role in understanding how we behave and how our bodies respond to infection.
The gut microbiome is made up of trillions of bacteria, fungi, and other microorganisms.
Each of these microorganisms can play an important role in our health, and it’s this community of bacteria and other microbes that can make us sick, so the microbiota can influence our immune systems.
So far, our understanding of how the gut microbiome affects the disease has been fairly limited, but our understanding is beginning to change.
TLRNPs are the molecules that control the response to the infection by a specific species of bacteria.
We know that there are TLRs that are expressed in the gut, and that the bacteria in our gut also have these TLRs, but we don’t know why these TLR-regulated responses are expressed.
TLRTs are thought to play important roles in regulating the immune response, and we are beginning to understand how these TLRNAs are expressed and how they are linked to disease.
TLTGs are proteins that are made up by a small group of microbes.
They are expressed on the surface of the cell surface and in the cells lining the gut.
TLRNPs control how the immune cells respond to the microbial community.
TLRNA are released by some types of bacteria that are important in our immune response.
TLTLRs are released when the immune cell has to respond to an infection by making a certain type of protein.
TL TGs are made by a different type of bacteria called TL Tregs.
TLG are released at the time the immune network is primed by an infection